Determination of nerve conduction abnormalities in patients with impaired glucose tolerance

Basit Öğe Kaydı
dc.authorid0000-0003-2016-9965en_US
dc.authorid0000-0003-2016-9965en_US
dc.authorid0000-0002-2887-9235en_US
dc.contributor.authorSahin, Sevki
dc.contributor.authorKarsidag, Sibel
dc.contributor.authorAyalp, Sunay
dc.contributor.authorSengul, Ahmet
dc.contributor.authorUs, Onder
dc.contributor.authorKarsidag, Kubilay
dc.date.accessioned2024-07-12T21:52:27Z
dc.date.available2024-07-12T21:52:27Z
dc.date.issued2009en_US
dc.departmentMaltepe Üniversitesien_US
dc.description.abstractRecent studies have shown that impaired glucose tolerance (IGT) is associated with dysfunction in the peripheral and autonomic nerves. The aim of this study was to determine the electrophysiological abnormalities of IGT. To determine electrophysiological abnormality in the large sensorimotor and sudomotor autonomic nerves with IGT patients, 43 patients and 34 healthy subjects have been studied. Subjective neuropathy symptoms, neurological examination and the electrophysiological findings were evaluated. When conduction of large somatic fibers only was evaluated, the ratio of electrophysiological abnormality was found to be 21%. In addition, where sympathetic skin response was evaluated the sudomotor autonomic abnormality ratio was 28% in upper extremities, 53% in lower extremities, and 16% in upper and lower extremities together. The percentages of abnormal electrophysiological parameters in different motor and sensory nerves were 39.5% in the peroneal motor nerve, 20.9% in the median motor and sural sensory nerves, 18.6% in the median sensory nerve, 16.3% in the tibial motor nerve, 14% in the ulnar sensory nerve, and 2.3% in the ulnar motor nerve. While distal motor latency was the most frequent abnormal parameter in the median and tibial motor nerves, the amplitude changes in the peroneal and ulnar motor nerves were also prominent. In sensory evaluation, the onset latency in the median-ulnar sensory nerves and the amplitude in the sural sensory nerve were found to be evident abnormalities.en_US
dc.identifier.doi10.1007/s10072-009-0089-8
dc.identifier.endpage289en_US
dc.identifier.issn1590-1874
dc.identifier.issue4en_US
dc.identifier.pmid19444381en_US
dc.identifier.scopus2-s2.0-67651006460en_US
dc.identifier.scopusqualityQ1en_US
dc.identifier.startpage281en_US
dc.identifier.urihttps://dx.doi.org/10.1007/s10072-009-0089-8
dc.identifier.urihttps://hdl.handle.net/20.500.12415/8373
dc.identifier.volume30en_US
dc.identifier.wosWOS:000267827700002en_US
dc.identifier.wosqualityQ4en_US
dc.indekslendigikaynakWeb of Science
dc.indekslendigikaynakScopus
dc.indekslendigikaynakPubMed
dc.language.isoenen_US
dc.publisherSPRINGERen_US
dc.relation.ispartofNEUROLOGICAL SCIENCESen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.snmzKY03096
dc.subjectImpaired glucose toleranceen_US
dc.subjectNerve conductionen_US
dc.subjectElectrophysiologicalen_US
dc.subjectAbnormalityen_US
dc.titleDetermination of nerve conduction abnormalities in patients with impaired glucose toleranceen_US
dc.typeArticle
dspace.entity.typePublication

Dosyalar

Koleksiyon

WoS İndeksli Yayınlar Koleksiyonu
PubMed İndeksli Yayınlar Koleksiyonu
Scopus İndeksli Yayınlar Koleksiyonu